On a basic level, fat acts as a receptacle to store energy, but upon a closer look it is an essential actor in vital bodily processes like the immune response, the regulation of insulin sensitivity, and maintenance of body temperature. Obesity is known to cause cardiometabolic diseases like hypertension and diabetes but attributing these diseases to merely an overabundance of fat is a simplification. The researchers highlight the potential of altering fat tissue phenotypes for therapeutic benefits, pointing to an exciting avenue for future medical advancements. This decline leads to health issues such as insulin resistance and inflammation. As fat loses its plasticity due to aging and obesity, it can’t respond to bodily cues, leading to insulin resistance, inflammation, and cell death due to the fast growth of adipose tissue that outstrips its blood supply.Ī review in the Cell journal reveals that the negative health impacts of obesity are not merely due to excess fat, but rather a loss of fat’s plasticity, its ability to respond to changes. The researchers explain that fat, besides storing energy, plays crucial roles in immune response, insulin sensitivity regulation, and body temperature maintenance. Obesity’s detrimental health effects, such as hypertension and diabetes, stem not merely from an excess of fat, but rather from the loss of fat’s plasticity - its ability to respond to changes - according to a review published in the Cell journal.
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